Type 2 diabetes has been treated primarily with medications that aim to reduce insulin levels in the blood.

Insulin is not the cause of type 2 diabetes, but rather a response to the underlying problem: insulin resistance.

Treating type 2 diabetes with medications that only aim to reduce insulin only masks the problem and perpetuates the cycle of insulin resistance.

Insulin is a key hormone in regulating blood sugar. Under normal conditions, when we eat, carbohydrates are broken down into glucose, causing a spike in blood sugar levels. Insulin helps cells absorb glucose and use it for energy.

However, in type 2 diabetes, the body’s cells become resistant to insulin, meaning insulin can no longer do its job efficiently. As a result, the pancreas produces more and more insulin in an attempt to compensate for the resistance, resulting in elevated insulin levels in the blood (hyperinsulinemia).

Traditional treatments for type 2 diabetes often focus on lowering insulin levels, either through oral medications like metformin or insulin injections. The logic is that by lowering insulin levels, blood glucose levels also decrease.

However, this does not address the underlying cause, which is insulin resistance, and in many cases, it can even worsen it in the long term. By lowering insulin without addressing the resistance in the cells, the body continues to produce more insulin, perpetuating the cycle of hyperinsulinemia and insulin resistance.

Insulin resistance is the primary problem in type 2 diabetes, not a lack of insulin.
Addressing insulin resistance through dietary changes and exercise is more effective in the long term for treating type 2 diabetes.

Today, insulin resistance in the early stages (and even in patients already with type 2 diabetes, which can be reversed) can be treated and completely reversed with dietary changes and proper supplementation.